Abstract Description
Neurophysiological mechanisms related to corticomotor excitability and neurochemical mechanisms linked to GABA activity may explain the analgesic response to prolonged continuous theta burst stimulation (pcTBS) targeted at the primary motor cortex (M1) and dorsolateral prefrontal cortex (DLPFC) in patients with neuropathic pain (NP) .
Presenters
Authors
Authors
Bhushan Thakkar - Virginia Commonwealth University and University of Dundee (Virginia, United States of America and United Kingdom) , Carrie Peterson - Virginia Comonwealth University (Virginia, United States of America) , Edmund Acevedo - Virginia Comonwealth University (Virginia , United States of America)
